Up Learn – A Level Psychology (AQA) – SCHIZOPHRENIA
Schizophrenia: Interactionist Approach
The interactionist explanation of schizophrenia says that schizophrenia is caused by an interaction between biological factors and psychological factors
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Schizophrenia
2. Diagnosing Schizophrenia (free trial)
3. Diagnosing Schizophrenia: Reliability (free trial)
4. Diagnosing Schizophrenia: Validity (free trial)
5. Reliability of Schizophrenia Diagnosis: Inter-Rater Reliability (free trial)
6. Validity of Schizophrenia Diagnosis: Rosenhan’s Study (free trial)
7. Cultural Bias in Schizophrenia Diagnosis (free trial)
8. Cultural Bias: Study Support (free trial)
9. Schizophrenia – Gender Bias (free trial)
10. Gender Bias: Study Support (free trial)
11. Diagnosing Schizophrenia: Comorbidity (free trial)
12. Effects of Comorbidity on Reliability and Validity (free trial)
13. Comorbidity in Schizophrenia: Study Support (free trial)
14. Symptom Overlap in Diagnostic Criteria (free trial)
15. Symptom Overlap: Study Support (free trial)
2. Genetic Explanations of Schizophrenia (free trial)
3. The Genetic Explanation of Schizophrenia: Twin Studies (free trial)
4. The Genetic Explanation of Schizophrenia: Limitations of Twin Studies (free trial)
5. The Genetic Explanation of Schizophrenia: Adoption Studies (free trial)
6. The Genetic Explanation of Schizophrenia: Limitations of Adoption Studies (free trial)
7. Brain Abnormalities and Schizophrenia (free trial)
8. The Neural Correlates Hypothesis (free trial)
9. Neural Correlates: Study Support (free trial)
10. Effects of Medication on Brain Structure (free trial)
11. Individual Differences in Brain Abnormalities (free trial)
12. The Dopamine Hypothesis (free trial)
13. The Revised Dopamine Hypothesis (free trial)
14. The Dopamine Hypothesis: Study Support (free trial)
15. Limitations of the Dopamine Hypothesis – Part 1 (free trial)
16. Limitations of the Dopamine Hypothesis – Part 2
17. Biological Treatments for Schizophrenia: Antipsychotic Drugs (free trial)
18. How Antipsychotic Drugs Work (free trial)
19. Limitations of Traditional Antipsychotics: Side Effects (free trial)
20. Limitations of Traditional Antipsychotics: Negative Symptoms (free trial)
21. Typical and Atypical Antipsychotics (free trial)
22. Support for Atypical Antipsychotics (free trial)
23. Comparing Effects of Atypical Antipsychotics and Typical Antipsychotics (free trial)
24. Limitations of Atypical Antipsychotics: Effectiveness (free trial)
2. The Cognitive Explanation of Schizophrenia: Dysfunctional Attention (free trial)
3. The Cognitive Explanation of Schizophrenia: Biased Reasoning (free trial)
4. Dysfunctional Thought Processes Lead to Delusions (free trial)
5. Dysfunctional Thought Processes Lead to Hallucinations (free trial)
6. Dysfunctional Thought Processes Lead to Negative Symptoms (free trial)
7. Study Support for the Cognitive Explanation (free trial)
8. Limitations of the Cognitive Explanation: Ignores Biological Factors (free trial)
9. Cognitive Treatments: Cognitive Behavioural Therapy (free trial)
10. Study Support for CBT (free trial)
11. Limitations of CBT: Study Limitations (free trial)
12. Limitations of CBT: Availability and Cost (free trial)
2. The Family Dysfunction Explanation of Schizophrenia (free trial)
3. Family Dysfunction: Double Bind (free trial)
4. Family Dysfunction: Expressed Emotion (free trial)
5. Family Dysfunction: Study Support (free trial)
6. Limitations of Family Dysfunction: Unreliable Evidence (free trial)
7. Limitations of Family Dysfunction: Individual Differences (free trial)
8. Family Therapy (free trial)
9. Study Support: Pharoah et al (free trial)
10. Support for Family Therapy: Cost Effectiveness (free trial)
11. Family Therapy: Limitations of Study Support (free trial)
Last time, we saw study support for the diathesis-stress model:
Tienari et al conducted an adoption study comparing kids whose biological mums had schizophrenia to a control group of adopted children whose biological mums didn’t have schizophrenia.
The children whose biological mums had schizophrenia were more likely to develop the disorder, meaning that gene alleles that they inherited from their mum were a diathesis for schizophrenia.
But, the child’s risk of developing schizophrenia also depended on the environment they grew up in: children only developed schizophrenia if they experienced stress in their adopted family!
This supports the idea that schizophrenia requires both a biological diathesis and an environmental stress to trigger the disorder.
Now, further support for the interactionist explanation comes from a study conducted by Brown and Birley in 1968…
In their study, Brown and Birley investigated individuals who had recently developed schizophrenia…
Brown and Birley conducted interviews with the patients and their families, asking them about the events that had occurred in the past 3 weeks before they had developed schizophrenia.
They found that, in the 3 weeks before the patients developed schizophrenia, 50% of them had experienced a major stressful life event…
Such as the death of a relative, or a new stressful job…
This supports the idea that stress acts as a trigger for the development of schizophrenia.
To sum up, we’ve now seen further support for the interactionist explanation of schizophrenia…
We’ve now seen further support for the interactionist explanation of schizophrenia, from a study by Brown and Birley.
Brown and Birley interviewed patients who had recently developed schizophrenia.
50% of them had a stressful life event in the 3 weeks prior to symptoms starting…
Which supports the idea that stress triggers schizophrenia.